Alcohol abuse (when defined as more than 21 standard drinks in one week) is already the second largest risk factor for the development of oral cancer. Alcohol's effect on the mouth may be the key to understanding how it works with tobacco to increase the risk of developing cancer.
The dehydrating effect of alcohol on cell walls enhances the ability of tobacco carcinogens to infuse mouth tissues; additionally, nutritional deficiencies associated with heavy drinking can lower the body's natural ability to use antioxidants to prevent the formation of cancers.
A major difficulty in the study of tobacco and alcohol as risk factors is that most oral cancer patients have used both products. Further research is necessary to determine the relationship between oral cancer, alcohol use and tobacco use.
However, it is widely accepted that eliminating the use of oral tobacco, and reducing or eliminating your intake of alcohol, will immediately reduce your risk of developing oral cancer. In fact, within 10 years your risk for oral cancer should be as low as any other non-drinker/non-smoker.
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